<ExportAllData>
  <Microbiota.Analysis>
    <AnalysisID>48</AnalysisID>
    <UUID>6597cd60-ab39-4eae-8311-c40102d8cb87</UUID>
    <MetaAnalysisID>2018_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1186/s40168-018-0416-5</DOI>
    <Title>Differential human gut microbiome assemblages during soil-transmitted helminth infections in Indonesia and Liberia</Title>
    <JournalID>2</JournalID>
    <Year>2018</Year>
    <Authors>Rosa MA, Supali T, Gankpala L, Djuardi Y, Sartono E, Zhou Y, Fischer K, Martin J, Tyagi R, Bolay FK, Fischer PU, Yazdanbakhsh M, Mitreva M</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA407815</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated conserved microbial signatures positively or negatively associated with soil-transmitted helminth infections across Liberia and Indonesia; in addition, longitudinal samples analysis from a double-blind randomized trial showed that the gut microbiota responds to deworming but does not transition closer to the uninfected state. The main compositional alterations detected in the faecal microbiota of infected individuals were as follows:
- Succinivibrio expanded in all helminth-infected individuals, irrespective of species of infecting parasites; 
- Solobacterium expanded in Necator and Trichuris-infected individuals 
- Desulfovibrio expanded in Ascaris and Trichuris-infected individuals 
- Four Firmicutes and one unclassified Bacteroidetes, including Allobaculum, expanded in Ascaris and Necator-infected individuals 
- Lachnospiraceae negatively associated with infection by Ascaris and Necator 
- Rhodococcus negatively associated with infection by Ascaris and Trichuris 
- Olsenella, Flavonifractor, and Enterococcus, Erysipelotrichaceae expanded in all infected individuals 
- Clostridium IV and Collinsella positively associated with infection in individuals from Liberia (but not from Indonesia).

Anthelmintic treatment resulted in expanded Enterococcus, Pseudomonadales, Flavobacteriaceae, Sphingobacteriaceae and reduced Ochrobactrum.
Deworming resulted in expanded  Sphingobacterium, Deltaproteobacteria, Erysipelotrichaceae and reduced Prevotellaceae, Sutterellaceae, Leuconostocaceae and Butyricimonas. 
Self-clearance resulted in expanded Bacteroidales families, with Turicibacter negatively associated with baseline infection.
Akkermansia and its parent phylogeny, as well as Ruminococcus, significantly associated with incomplete worm clearance or reinfection.</PredominantChangesReported>
    <taxa_link_Family>14,18</taxa_link_Family>
    <taxa_link_Genus>8,29,32,37,42,74,89,104</taxa_link_Genus>
    <taxa_link_Phylum>3,9</taxa_link_Phylum>
    <_link_ParasiteSpecies>19,9,3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-18T15:20:05.78+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T13:03:17.37+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>49</AnalysisID>
    <UUID>8cde7e67-b9f0-44dc-9aba-04357e54b87f</UUID>
    <MetaAnalysisID>2018_2</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/s41598-018-30412-x </DOI>
    <Title>Schistosoma mansoni infection is associated with quantitative and qualitative modi cations of the mammalian intestinal microbiota</Title>
    <JournalID>22</JournalID>
    <Year>2018</Year>
    <Authors>Timothy P. Jenkins, Laura E. Peachey, Nadim J. Ajami, Andrew S. MacDonald, Michael H. Hsieh, Paul J. Brindley, Cinzia Cantacessi, Gabriel Rinaldi</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>3</DBArchiveID>
    <ProjectNumber>10.17632/y8c7vpc8zp.1</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>↓Alpha diversity, ↑beta diversity</PredominantChangesReported>
    <taxa_link_Class>11,21</taxa_link_Class>
    <taxa_link_Family>29</taxa_link_Family>
    <taxa_link_Genus>13</taxa_link_Genus>
    <taxa_link_Order>2,6,15,23,24</taxa_link_Order>
    <taxa_link_Phylum>16</taxa_link_Phylum>
    <taxa_link_Species>1,2</taxa_link_Species>
    <_link_ParasiteSpecies>20</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>8</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-18T15:32:43.65+00:00</CreatedOn>
    <Active>false</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>50</AnalysisID>
    <UUID>3a7de6e6-d4b7-48fc-b93d-1d01f1751741</UUID>
    <MetaAnalysisID>2010_1</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>64</ProfilingMethodID>
    <DOI>https://doi.org/10.1002/ibd.21299</DOI>
    <Title>Alteration of the murine gut microbiota during infection with the parasitic helminth Heligmosomoides polygyrus</Title>
    <JournalID>10</JournalID>
    <Year>2010</Year>
    <Authors>Walk ST, Blum AM, Ang-Sheng S, Weinstock JV, Young VB  </Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>N/A (qPCR)</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated changes in the composition of the ileal and coecal microbiota of C57BL/6 mice infected with Heligmosomoides polygyrus, using samples collected 2 weeks post-experimental infection.</PredominantChangesReported>
    <taxa_link_Family>6,14,19</taxa_link_Family>
    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2,2048</_link_SampleSite>
    <_link_SampleType>16</_link_SampleType>
    <CreatedOn>2019-06-18T19:10:45.82+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T15:52:22.74+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>51</AnalysisID>
    <UUID>e1ce85ff-febb-4600-9b14-7e7f21294216</UUID>
    <MetaAnalysisID>2011_1</MetaAnalysisID>
    <HostSpeciesID_Control>2</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0024417</DOI>
    <Title>Metagenome plasticity of the bovine abomasal microbiota in immune animals in response to Ostertagia ostertagi Infection</Title>
    <JournalID>20</JournalID>
    <Year>2011</Year>
    <Authors>Li RW, Wu S, Li W, Huang Y, Gasbarre LC</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA518072</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterized the abomasal microbiota composition of cattle partially immune to Ostertagia ostertagi infection in response to reinfection. </PredominantChangesReported>
    <taxa_link_Genus>46,102</taxa_link_Genus>
    <_link_ParasiteSpecies>2</_link_ParasiteSpecies>
    <_link_InfectedSpecies>2</_link_InfectedSpecies>
    <_link_SampleSite>16</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-18T19:16:35.35+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T15:49:31.68+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>52</AnalysisID>
    <UUID>8b7c0e8c-2f20-4ed3-8415-cf1d9fc1d970</UUID>
    <MetaAnalysisID>2012_1</MetaAnalysisID>
    <HostSpeciesID_Control>3</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.ppat.1003000</DOI>
    <Title>Therapeutic helminth infection of macaques with idiopathic chronic diarrhea alters the inflammatory signature and mucosal microbiota of the colon</Title>
    <JournalID>19</JournalID>
    <Year>2012</Year>
    <Authors>Broadhurst MJ, Ardeshir A, Kanwar B, Mirpuri J, Gundra UM, Leung JM, Wiens KE, Vujkovic-Cvijin I, Kim CC, Yarovinsky F, Lerche NW, McCune JM, Loke P</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>N/A</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>The study investigated the effects of experimental infection of captive rhesus monkeys with Trichuris trichiura on the composition of microbial communities attached to the intestinal mucosa, and established correlations between such changes and the amelioration of clinical signs of idiopathic chronic diarrhoea.  </PredominantChangesReported>
    <taxa_link_Family>2,11,29,36</taxa_link_Family>
    <taxa_link_Genus>13,83,92</taxa_link_Genus>
    <taxa_link_Order>2,5,8</taxa_link_Order>
    <taxa_link_Phylum>3,15</taxa_link_Phylum>
    <_link_ParasiteSpecies>3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>3</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <_link_SampleType>64,256</_link_SampleType>
    <CreatedOn>2019-06-18T19:23:33.32+00:00</CreatedOn>
    <UpdatedOn>2019-07-05T09:35:45.26+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>53</AnalysisID>
    <UUID>29333887-5ac9-48eb-bb3d-d6436ffbbac8</UUID>
    <MetaAnalysisID>2012_2</MetaAnalysisID>
    <HostSpeciesID_Control>4</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>16</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0035470</DOI>
    <Title>Worm burden-dependent disruption of the porcine colon microbiota by Trichuris suis infection</Title>
    <JournalID>20</JournalID>
    <Year>2012</Year>
    <Authors>Wu S, Li RW, Li W, Beshah E, Dawson HD, Urban JF Jr</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>4</DBArchiveID>
    <ProjectNumber>4474250.3 to 4474257.3, 4474259.3, 4474261.3 and 4474262.3</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>
This study characterised the microbiota composition of the porcine proximal colon in response to Trichuris suis infection, 53 days post-parasite inoculation.</PredominantChangesReported>
    <taxa_link_Genus>4,19,22,39,48,49,72,92,98,107,108</taxa_link_Genus>
    <taxa_link_Phylum>8,11,14,15</taxa_link_Phylum>
    <_link_ParasiteSpecies>4</_link_ParasiteSpecies>
    <_link_InfectedSpecies>4</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-18T19:30:29.71+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T15:43:37.8+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>54</AnalysisID>
    <UUID>1e9b8ff7-6e52-46d8-ab05-523e42fc7155</UUID>
    <MetaAnalysisID>2012_3</MetaAnalysisID>
    <HostSpeciesID_Control>4</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>64</ProfilingMethodID>
    <DOI>https://doi.org/10.1128/IAI.00141-12</DOI>
    <Title>Alterations in the porcine colon microbiota induced by the gastrointestinal nematode Trichuris suis</Title>
    <JournalID>1</JournalID>
    <Year>2012</Year>
    <Authors>Li RW, Wu S, Li W, Navarro K, Couch RD, Hill D, Urban JF Jr.</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA518074</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised the microbiota composition of  the porcine proximal colon in response to Trichuris suis infection, 21 days post-parasite inoculation.</PredominantChangesReported>
    <taxa_link_Genus>11,18,33,37,39,47,70,75,76,81,83,92,94,95,98,99,104</taxa_link_Genus>
    <taxa_link_Phylum>5</taxa_link_Phylum>
    <_link_ParasiteSpecies>4</_link_ParasiteSpecies>
    <_link_InfectedSpecies>4</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-18T19:44:24.7+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T11:54:21+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>55</AnalysisID>
    <UUID>492a1395-4be4-4854-903b-df4c5eabcbba</UUID>
    <MetaAnalysisID>2013_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0076573</DOI>
    <Title>Patent human infections with the whipworm, Trichuris trichiura, are not associated with alterations in the faecal microbiota</Title>
    <JournalID>20</JournalID>
    <Year>2013</Year>
    <Authors>Cooper P, Walker AW, Reyes J, Chico M, Salter SJ, Vaca M, Parkhill J</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP002465</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised the faecal microbiota of 97 children, ie. 30 uninfected, 17 infected with Trichuris trichiura, and 50 with T. trichiura and Ascaris lumbricoides from Ecuador. Post-treatment samples were analyzed for 14 children initially infected with T. trichiura alone and for 21 uninfected children. Anthelmintic treatment of children with T. trichiura did not alter faecal microbiota composition.</PredominantChangesReported>
    <taxa_link_Genus>29,101</taxa_link_Genus>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <CreatedOn>2019-06-20T09:41:55.75+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T15:37:49.19+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>56</AnalysisID>
    <UUID>bf7455f2-2345-4312-87e2-cea880968e3a</UUID>
    <MetaAnalysisID>2013_2</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>128</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0074026</DOI>
    <Title>Small intestinal nematode infection of mice is associated with increased enterobacterial loads alongside the intestinal tract</Title>
    <JournalID>20</JournalID>
    <Year>2013</Year>
    <Authors>Rausch S, Held J, Fischer A, Heimesaat MM, Kühl AA, Bereswill S, Hartmann S</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>N/A</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated intestinal microbiota changes in the ileum, cecum and colon of C57BL/6 mice infected my Heligmosomoides polygyrus at 14 days post-nematode infection. The altered microbiota composition was independent of the IL-4/-13 - STAT6 signaling axis, as infected IL-4Rα(-/-) mice showed a similar increase in enterobacterial loads.</PredominantChangesReported>
    <taxa_link_Class>14</taxa_link_Class>
    <taxa_link_Family>12</taxa_link_Family>
    <taxa_link_Genus>13,58</taxa_link_Genus>
    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2,128,2048</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-20T09:48:10.19+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T15:34:52.69+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>57</AnalysisID>
    <UUID>05e5beb4-6d63-428b-9f6e-3d75fc24d876</UUID>
    <MetaAnalysisID>2013_3</MetaAnalysisID>
    <HostSpeciesID_Control>7</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1096/fj.13-232751</DOI>
    <Title>Infection with the carcinogenic liver fluke Opisthorchis viverrini modifies intestinal and biliary microbiome</Title>
    <JournalID>23</JournalID>
    <Year>2013</Year>
    <Authors>Plieskatt JL, Deenonpoe R, Mulvenna JP, Krause L, Sripa B, Bethony JM, Brindley PJ</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA188112</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised differences in microbiota composition of bile and colorectal contents of Syrian golden hamsters 6 weeks post-experimental infection by Opisthorchis viverrini. </PredominantChangesReported>
    <taxa_link_Family>14,16,18,19,32,36</taxa_link_Family>
    <taxa_link_Genus>1,58,79,85</taxa_link_Genus>
    <taxa_link_Phylum>14</taxa_link_Phylum>
    <_link_ParasiteSpecies>7</_link_ParasiteSpecies>
    <_link_InfectedSpecies>7</_link_InfectedSpecies>
    <_link_SampleSite>512,1024</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-20T09:52:19.4+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T11:57:29.39+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>58</AnalysisID>
    <UUID>cd06c96f-8f54-44f5-8b91-33b68e983bde</UUID>
    <MetaAnalysisID>2014_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pntd.0002880</DOI>
    <Title>Helminth colonization is associated with increased diversity of the gut microbiota</Title>
    <JournalID>18</JournalID>
    <Year>2014</Year>
    <Authors>Lee SC, Tang MS, Lim YAL, Choy SH, Kurtz ZD, Cox LM, Gundra UM, Cho I, Bonneau R, Blaser MJ, Chua KH, Loke P</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>N/A</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This compared the composition and diversity of bacterial communities from the fecal microbiota of 51 people from two villages in Malaysia, of which 36 were infected by helminths. Helminth-colonized individuals had greater species richness and number of observed OTUs with enrichment of Paraprevotellaceae, especially with Trichuris infection. </PredominantChangesReported>
    <taxa_link_Class>2,17,20</taxa_link_Class>
    <taxa_link_Family>3,27,37</taxa_link_Family>
    <taxa_link_Genus>17,25</taxa_link_Genus>
    <taxa_link_Order>2,3,19</taxa_link_Order>
    <_link_ParasiteSpecies>8</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T09:58:11.98+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T11:59:22.47+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>59</AnalysisID>
    <UUID>0dd86c44-3c80-428d-bfbc-69910745138c</UUID>
    <MetaAnalysisID>2014_2</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1093/infdis/jiu256</DOI>
    <Title>Impact of experimental hookworm infection on the human gut microbiota</Title>
    <JournalID>24</JournalID>
    <Year>2014</Year>
    <Authors>Cantacessi C, Giacomin P, Croese J, Zakrzewski M, Sotillo J, McCann L, Nolan MJ, Mitreva M, Krause L, Loukas A</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>SRP041283</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterized the differences in composition and relative abundance of fecal microbial communities in human subjects prior to and 8 weeks following experimental infection with Necator americanus. </PredominantChangesReported>
    <_link_ParasiteSpecies>9</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T10:02:52.23+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:54:06.32+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>60</AnalysisID>
    <UUID>18b0e7d7-4506-41d5-820c-905bfb50f73d</UUID>
    <MetaAnalysisID>2014_3</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>64</ProfilingMethodID>
    <DOI>https://doi.org/10.4161/gmic.32155</DOI>
    <Title>Commensal–pathogen interactions in the intestinal tract: Lactobacilli promote infection with, and are promoted by, helminth parasites</Title>
    <JournalID>9</JournalID>
    <Year>2014</Year>
    <Authors>Reynolds LA, Smith KA, Filbey KJ, Harcus Y, Hewitson JP, Redpath SA, Valdez Y, Yebra MJ, Finlay BB, Maizels RM</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>N/A (clone library)</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study showed that abundance of Lactobacillus bacteria correlates positively with infection with Heligmosomoides polygyrus, as well as with heightened regulatory T cell (Treg) and Th17 responses. H. polygyrus infection raised Lactobacillus species abundance in the duodenum of susceptible C57BL/6 mice, but not in relatively resistant BALB/c mice. Experimental administration of Lactobacillus taiwanensis to BALB/c mice elevates regulatory T cell frequencies and results in greater helminth establishment. </PredominantChangesReported>
    <taxa_link_Family>12,19</taxa_link_Family>
    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>32</_link_SampleSite>
    <_link_SampleType>2,8</_link_SampleType>
    <CreatedOn>2019-06-20T10:06:41.97+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:49:00.34+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>61</AnalysisID>
    <UUID>9e8949ea-c9ce-47bb-b94d-86aa4a31a8bb</UUID>
    <MetaAnalysisID>2015_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/srep13797</DOI>
    <Title>Experimental hookworm infection and escalating gluten challenges are associated with increased microbial richness in celiac subjects</Title>
    <JournalID>22</JournalID>
    <Year>2015</Year>
    <Authors>Giacomin P, Zakrzewski M, Croese J, Su X, Sotillo J, McCann L, Navarro S, Mitreva M, Krause L, Loukas A, Cantacessi C</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>SRP059769</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study assessed the qualitative and quantitative changes in the microbiota of 8 human volunteers with coeliac disease prior to and following infection with Necator americanus, and 24, 36 and 52 weeks following challenge with escalating doses of dietary gluten. </PredominantChangesReported>
    <_link_ParasiteSpecies>9</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T10:11:06.69+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:45:03.97+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>62</AnalysisID>
    <UUID>74970d66-c899-4e02-aeb8-80b8ad36e04c</UUID>
    <MetaAnalysisID>2015_2</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0125495</DOI>
    <Title>Chronic Trichuris muris infection decreases diversity of the intestinal microbiota and concomitantly increases the abundance of Lactobacilli</Title>
    <JournalID>20</JournalID>
    <Year>2015</Year>
    <Authors>Holm JB, Sorobetea D, Kiilerich P, Ramayo-Caldas Y, Estellé J, Ma T, Madsen L, Kristiansen K, Svensson-Frej M  </Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP006108</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study assessed the influence of low-dose chronic infection of C57BL/6 mice with Trichuris muris, using faecal samples collected at 13, 20 and 27 days post-infection, as well as luminal colon content samples collected at 35 days post-infection.  </PredominantChangesReported>
    <taxa_link_Class>3,10</taxa_link_Class>
    <taxa_link_Family>14,19</taxa_link_Family>
    <taxa_link_Genus>8,15,21,54,58,76,82,91,97</taxa_link_Genus>
    <taxa_link_Order>10,15</taxa_link_Order>
    <taxa_link_Phylum>0</taxa_link_Phylum>
    <taxa_link_Species>0</taxa_link_Species>
    <_link_ParasiteSpecies>10</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <_link_SampleType>2,8</_link_SampleType>
    <CreatedOn>2019-06-20T10:24:22.11+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:03:40.1+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>63</AnalysisID>
    <UUID>254fd3c6-1d67-469a-98d6-729b3c29dc37</UUID>
    <MetaAnalysisID>2015_3</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0125945</DOI>
    <Title>Chronic Trichuris muris infection in C57BL/6 mice causes significant changes in host microbiota and metabolome: effects reversed by pathogen clearance</Title>
    <JournalID>20</JournalID>
    <Year>2015</Year>
    <Authors>Houlden A, Hayes KS, Bancroft AJ, Worthington JJ, Wang P, Grencis RK, Roberts IS </Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP008663</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated gut microbial communities of C57BL/6 mice infected by Trichuris muris profiled using DGGE, 454 pyrosequencing, and metabolomics. Changes in microbial composition occurred between 14 and 28 days post infection, resulting in significant changes in α and β- diversity. This impact was dominated by a reduction in the diversity and abundance of Bacteroidetes, specifically Prevotella and Parabacteroides. Metabolomic analysis of stool samples of infected mice at day 41 showed significant differences to uninfected controls with a significant increase in the levels of a number of essential amino acids and a reduction in breakdown of dietary plant derived carbohydrates. Following clearance of infection the intestinal microbiota underwent additional changes gradually transitioning by day 91 towards a microbiota of an uninfected animal. </PredominantChangesReported>
    <taxa_link_Genus>71,80,84</taxa_link_Genus>
    <taxa_link_Phylum>3</taxa_link_Phylum>
    <_link_ParasiteSpecies>10</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T10:29:14.49+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:31:57.99+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>64</AnalysisID>
    <UUID>30c9b0b3-888f-4676-b5b7-fb5b644515fa</UUID>
    <MetaAnalysisID>2015_4</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1186/s40168-015-0103-8</DOI>
    <Title>Type 2 immunity-dependent reduction of segmented filamentous bacteria in mice infected with the helminthic parasite Nippostrongylus brasiliensis</Title>
    <JournalID>2</JournalID>
    <Year>2015</Year>
    <Authors>Fricke WF, Song Y, Wang AJ, Smith A, Grinchuk V, Pei C, Ma B, Lu N, Urban JF Jr, Shea-Donohue T, Zhao A</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA255974</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study demonstrated that the effects of Nippostrongylus braziliensis infection on the composition of the murine gut microbiota, host antimicrobial proteins (AMP) and IL-17 expression, were attenuated in STAT6 -/- and IL-13 -/- knockout mice.</PredominantChangesReported>
    <taxa_link_Family>6,8,19,30,37,39</taxa_link_Family>
    <taxa_link_Phylum>1,2,9</taxa_link_Phylum>
    <taxa_link_Species>3</taxa_link_Species>
    <_link_ParasiteSpecies>11</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>64</_link_SampleSite>
    <_link_SampleType>2,8</_link_SampleType>
    <CreatedOn>2019-06-20T10:33:35.32+00:00</CreatedOn>
    <UpdatedOn>2019-07-05T09:49:50.09+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>65</AnalysisID>
    <UUID>dfd433aa-c09c-4403-8373-c272fc4c2dd3</UUID>
    <MetaAnalysisID>2015_5</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://10.1098/rstb.2014.0295</DOI>
    <Title>Interactions between multiple helminths and the gut microbiota in wild rodents</Title>
    <JournalID>17</JournalID>
    <Year>2015</Year>
    <Authors>Kreisinger J, Bastien G, Hauffe HC, Marchesi J, Perkins SE</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP009377</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This stud investigated the association between microbiota diversity and composition and natural infection of multiple helminth species in 29 wild mice (Apodemus flavicollis) infected by Heligmosomoides polygyrus, Syphacia spp. and Hymenolepis spp. Within this wild rodent system, variation in the composition and abundance of gut microbial taxa associated with helminths was specific to each helminth species and occurred both up- and downstream of a given helminth's niche. The most pronounced helminth–microbiota association was between the presence of tapeworms in the small intestine and increased S24–7 (Bacteroidetes) family in the stomach. </PredominantChangesReported>
    <taxa_link_Family>19,37</taxa_link_Family>
    <taxa_link_Phylum>2,9</taxa_link_Phylum>
    <_link_ParasiteSpecies>1,13,12</_link_ParasiteSpecies>
    <_link_InfectedSpecies>11</_link_InfectedSpecies>
    <_link_SampleSite>2,128,2048,256</_link_SampleSite>
    <_link_SampleType>8,16</_link_SampleType>
    <CreatedOn>2019-06-20T10:40:31.99+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:23:53.62+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>66</AnalysisID>
    <UUID>7809be47-46cd-470c-b8ae-e8511641f0c6</UUID>
    <MetaAnalysisID>2015_6</MetaAnalysisID>
    <HostSpeciesID_Control>12</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1080/19490976.2015.1047128</DOI>
    <Title>Alteration of the rat cecal microbiome during colonization with the helminth Hymenolepis diminuta</Title>
    <JournalID>9</JournalID>
    <Year>2015</Year>
    <Authors>McKenney EA, Williamson L, Yoder AD, Rawls JF, Bilbo SD, Parker W</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA270622</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, the microbiome of the cecum in Sprague Dawley rats colonized for 2 generations with the small intestinal helminth Hymenolepis diminuta was evaluated. </PredominantChangesReported>
    <taxa_link_Class>3,6</taxa_link_Class>
    <taxa_link_Family>30</taxa_link_Family>
    <taxa_link_Genus>110</taxa_link_Genus>
    <_link_ParasiteSpecies>13</_link_ParasiteSpecies>
    <_link_InfectedSpecies>12</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-20T10:45:59.35+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:07:54.55+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>67</AnalysisID>
    <UUID>978d08a7-610b-4cfe-a3e1-ae89db549561</UUID>
    <MetaAnalysisID>2015_7</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.ijpara.2015.04.001</DOI>
    <Title>(DOG) - Differences in the faecal microbiome of non-diarrhoeic clinically healthy dogs and cats associated with Giardia duodenalis infection: impact of hookworms and coccidia</Title>
    <JournalID>11</JournalID>
    <Year>2015</Year>
    <Authors>Šlapeta J, Dowd SE, Alanazi AD, Westman ME, Brown GK</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA276586</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study compared the composition and diversity of bacterial communities from 40 dogs, including free-roaming dogs, and 21 surrendered cats from Australia. The dog cohort included 17 (42.5%) dogs positive for Giardia and 13 (32.5%) dogs positive for dog hookworm (Ancylostoma caninum). The cat samples included eight positive for Giardia and eight positive for Cystoisospora. In dogs, significant difference in faecal microbial profiles (at genus level) were observed between the Giardia-positive and -negative groups. No such difference was demonstrated between Ancylostoma-positive and -negative dogs. </PredominantChangesReported>
    <taxa_link_Genus>13,18,24,29,51,55,86,87</taxa_link_Genus>
    <_link_ParasiteSpecies>142,31</_link_ParasiteSpecies>
    <_link_InfectedSpecies>24,13</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T10:49:43.8+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:12:40.52+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>68</AnalysisID>
    <UUID>64fbff77-e1e7-4d63-a52e-bc206c68791a</UUID>
    <MetaAnalysisID>2016_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/srep36797</DOI>
    <Title>Changes in duodenal tissue-associated microbiota following hookworm infection and consecutive gluten challenges in humans with coeliac disease</Title>
    <JournalID>22</JournalID>
    <Year>2016</Year>
    <Authors>Giacomin P, Zakrzewski M, Jenkins TP, Su X, Al-Hallaf R, Croese J, de Vries S, Grant A, Mitreva M, Loukas A, Krause L, Cantacessi C</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>SRP078558</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the changes in gut microbiota microbiota composition at the site of infection by Necator americanus and gluten-dependent inflammation in 6 human volunteers with coeliac disease using biopsy tissue from the duodenum collected prior to infection, as well as 24 weeks post-infection (i.e. following the administration of 10–50 mg/day gluten from weeks 12–24) and 36 weeks post-infection (i.e. following the administration of twice-weekly 1 g/day gluten from weeks 24 to 36). </PredominantChangesReported>
    <taxa_link_Class>4,12</taxa_link_Class>
    <taxa_link_Family>33,42</taxa_link_Family>
    <taxa_link_Genus>84</taxa_link_Genus>
    <taxa_link_Order>2,11</taxa_link_Order>
    <_link_ParasiteSpecies>9</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleSite>32</_link_SampleSite>
    <_link_SampleType>64</_link_SampleType>
    <CreatedOn>2019-06-20T10:53:55.63+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:01:59.41+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>69</AnalysisID>
    <UUID>de777233-145b-482f-a1ce-dd412b9d6493</UUID>
    <MetaAnalysisID>2016_2</MetaAnalysisID>
    <HostSpeciesID_Control>14</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0159770</DOI>
    <Title>Impact of helminth infections and nutritional constraints on the small intestine microbiota</Title>
    <JournalID>20</JournalID>
    <Year>2016</Year>
    <Authors>Cattadori IM, Sebastian A, Hao H, Katani R, Albert I, Eilertson KE, Kapur V, Pathak A, Mitchell S</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study used the Trichostrongylus retortaeformis-rabbit system to examine how the helminth infection and host restriction from coprophagy/ready-to-absorb nutrients affected the duodenal microbiota. Helminths reduced the diversity and abundance of the microbiota while the combination of parasites and coprophagic restriction led to a more diversified and abundant microbiota than infected cases, without significantly affecting the intensity of infection. Animals restricted from coprophagy and free from parasites exhibited the richest and most abundant bacterial community.  </PredominantChangesReported>
    <taxa_link_Family>2,9,22,31,36</taxa_link_Family>
    <taxa_link_Genus>13,36,61,92</taxa_link_Genus>
    <taxa_link_Phylum>9,13,14</taxa_link_Phylum>
    <_link_ParasiteSpecies>15</_link_ParasiteSpecies>
    <_link_InfectedSpecies>14</_link_InfectedSpecies>
    <_link_SampleSite>32</_link_SampleSite>
    <_link_SampleType>16</_link_SampleType>
    <CreatedOn>2019-06-20T10:58:36.22+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T13:42:10.2+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>70</AnalysisID>
    <UUID>c8980ecc-3e4c-4064-abcc-e7fe74358925</UUID>
    <MetaAnalysisID>2016_3</MetaAnalysisID>
    <HostSpeciesID_Control>15</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/srep20606</DOI>
    <Title>The effect of helminth infection on the microbial composition and structure of the caprine abomasal microbiome</Title>
    <JournalID>22</JournalID>
    <Year>2016</Year>
    <Authors>Li RW, Li W, Sun J, Yu P, Baldwin RL, Urban JF Jr</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>4</DBArchiveID>
    <ProjectNumber>mgp13390; mgm4629311.3 to mgm4629350.3</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterized the impact of H. contortus infection on the abomasal microbiome of 14 parasite naive goats  inoculated with 5,000 H. contortus infective larvae and 6 age-matched uninfected controls, 50 days post-experimental infection. </PredominantChangesReported>
    <taxa_link_Family>18</taxa_link_Family>
    <taxa_link_Genus>103</taxa_link_Genus>
    <taxa_link_Order>17</taxa_link_Order>
    <taxa_link_Phylum>7</taxa_link_Phylum>
    <taxa_link_Species>9</taxa_link_Species>
    <_link_ParasiteSpecies>16</_link_ParasiteSpecies>
    <_link_InfectedSpecies>15</_link_InfectedSpecies>
    <_link_SampleSite>16</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-20T12:40:16.37+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:18:24.77+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>71</AnalysisID>
    <UUID>d042a19b-fb24-425f-80c6-2837bb95b2de</UUID>
    <MetaAnalysisID>2016_4</MetaAnalysisID>
    <HostSpeciesID_Control>13</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1186/s13071-016-1908-4</DOI>
    <Title>Helminth infections and gut microbiota – a feline perspective</Title>
    <JournalID>3</JournalID>
    <Year>2016</Year>
    <Authors>Duarte AM, Jenkins TP, Latrofa MS, Giannelli A, Papadopoulos E, Madeira de Carvalho L, Nolan MJ, Otranto D, Cantacessi C</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA349988</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated qualitative and quantitative differences in faecal microbial profiles between cats with patent infections by Toxocara cati (n = 24) and uninfected controls (n = 21).</PredominantChangesReported>
    <taxa_link_Class>7,14</taxa_link_Class>
    <taxa_link_Family>7,13</taxa_link_Family>
    <taxa_link_Genus>20,32,39,42,56,58,92</taxa_link_Genus>
    <taxa_link_Order>6,15</taxa_link_Order>
    <taxa_link_Phylum>1</taxa_link_Phylum>
    <_link_ParasiteSpecies>17</_link_ParasiteSpecies>
    <_link_InfectedSpecies>13</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T12:44:18.12+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:19:52.54+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>72</AnalysisID>
    <UUID>e897b9e1-445b-4ea2-9dde-a7911d312fdd</UUID>
    <MetaAnalysisID>2017_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0184719</DOI>
    <Title>Infections by human gastrointestinal helminths are associated with changes in faecal microbiota diversity and composition</Title>
    <JournalID>20</JournalID>
    <Year>2017</Year>
    <Authors>Jenkins TP, Rathnayaka Y, Perera PK, Peachey LE, Nolan MJ, Krause L, Rajakaruna RS, Cantacessi C </Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJEB21999</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study explored the qualitative and quantitative differences between the microbial community profiles of cohorts of human volunteers from Sri Lanka with patent infection by one or more parasitic nematode species (H+ = 11), as well as that of uninfected subjects (H- = 11) and of volunteers who had been subjected to regular prophylactic anthelmintic treatment (Ht = 11). No significant differences in alpha diversity (Shannon) and richness between groups were detected;  however, beta diversity was significantly increased in H+ and Ht when individually compared to H-volunteers. Among others, bacteria of the families Verrucomicrobiaceae and Enterobacteriaceae showed a trend towards increased abundance in H+, whereas the Leuconostocaceae and Bacteroidaceae showed a relative increase in H- and Ht respectively. </PredominantChangesReported>
    <taxa_link_Class>21</taxa_link_Class>
    <taxa_link_Family>2,12,23,41</taxa_link_Family>
    <taxa_link_Genus>7,13,60</taxa_link_Genus>
    <taxa_link_Order>24</taxa_link_Order>
    <taxa_link_Species>1</taxa_link_Species>
    <_link_ParasiteSpecies>18,19,9,3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T12:49:01.79+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:23:07.77+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>73</AnalysisID>
    <UUID>234a9b6d-cae9-42aa-8884-9580bfad0180</UUID>
    <MetaAnalysisID>2017_2</MetaAnalysisID>
    <HostSpeciesID_Control>12</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pone.0182205</DOI>
    <Title>A benign helminth alters the host immune system and the gut microbiota in a rat model system</Title>
    <JournalID>20</JournalID>
    <Year>2017</Year>
    <Authors>Wegener Parfrey L, Jirků M, Šíma R, Jalovecká M, Sak B, Grigore K, Jirků Pomajbíkova K</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP014631</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the impact of infection by Hymenolepis diminuta on the gut microbial composition of a rat model system, prior to infection as well as at 10, 15, 30, 40, 50, 60, 70 and 80 days post-infection. </PredominantChangesReported>
    <taxa_link_Class>17</taxa_link_Class>
    <taxa_link_Family>13,14,36</taxa_link_Family>
    <taxa_link_Genus>105,110</taxa_link_Genus>
    <_link_ParasiteSpecies>13</_link_ParasiteSpecies>
    <_link_InfectedSpecies>12</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T12:53:33.29+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:24:05.23+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>74</AnalysisID>
    <UUID>c2f19866-eaf5-4b19-a7d5-95247f60f183</UUID>
    <MetaAnalysisID>2018_2</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/s41598-018-30412-x </DOI>
    <Title>Schistosoma mansoni infection is associated with quantitative and qualitative modifcations of the mammalian intestinal microbiota</Title>
    <JournalID>22</JournalID>
    <Year>2018</Year>
    <Authors>Jenkins TP, Peachey LE, Ajami NJ, MacDonald AS, Hsieh MH, Brindley PJ, Cantacessi C, Rinaldi G</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>3</DBArchiveID>
    <ProjectNumber>10.17632/y8c7vpc8zp.1</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised the fluctuations in the composition of the gut microbial flora of the small and large intestine, as well as the changes in abundance of individual microbial species, of Swiss-Webster mice experimentally infected with Schistosoma mansoni, prior to and following the onset of egg laying (at 28 and 50 days post-infection, respectively). A significant expansion in populations of bacteria of the Family Verrucomicrobiaceae (species Akkermansia muciniphila) was detected at both intestinal sites and at both time-points in infected mice in comparison to uninfected controls. Bacteria belonging to the Lactobacillaceae were also expanded in the large intestine of schistosome-infected mice mice at 28 days p.i., while those belonging to the Orders Bacteroidales, Coriobacteriales and Clostridiales were expanded in either the small intestine, i.e. Bacteroides acidifaciens, Lachnospiraceae, and/or large intestine of infected mice at 50 days p.i., i.e. Bacteroides acidifaciens, Parabacteroides, Adlercreutzia, Lachnospiraceae, Oscillospira. Conversely, a marked contraction of bacteria of the Class Erysipelotrichia was evident at both small and large intestine of helminth-infected mice at day 50 p.i. compared to uninfected controls.  </PredominantChangesReported>
    <taxa_link_Class>11,21</taxa_link_Class>
    <taxa_link_Family>2,6,8,18,19,32,41</taxa_link_Family>
    <taxa_link_Genus>5,7,8,13,39,58,77,80,110</taxa_link_Genus>
    <taxa_link_Order>2,6,15,23,24</taxa_link_Order>
    <taxa_link_Phylum>16</taxa_link_Phylum>
    <taxa_link_Species>1,2,7,8</taxa_link_Species>
    <_link_ParasiteSpecies>20</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>8,64</_link_SampleSite>
    <_link_SampleType>8</_link_SampleType>
    <CreatedOn>2019-06-20T13:09:12.96+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:34:44.7+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>75</AnalysisID>
    <UUID>4d94b0a7-6449-4caa-b40d-48d9775ba024</UUID>
    <MetaAnalysisID>2018_3</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1186/s13071-018-2739-2</DOI>
    <Title>Investigations on the interplays between Schistosoma mansoni, praziquantel and the gut microbiome</Title>
    <JournalID>3</JournalID>
    <Year>2018</Year>
    <Authors>Schneeberger PHH, Coulibaly JT, Panic G, Daubenberger C, Gueuning M, Frey JE, Keiser J</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated differences in faecal microbiota composition between Schistosoma mansoni infected (n = 34) and uninfected children (n = 6) from Côte d'Ivoire. From each infected child one pre-treatment, one 24-hour and one 21-day follow-up sample after administration of 60 mg/kg praziquantel or placebo, were collected. The abundance of bacteria belonging to the order Fusobacteriales was significantly increased in the gut microbiota of cured individuals, both at baseline and 24 hours post-treatment.</PredominantChangesReported>
    <taxa_link_Class>13,19</taxa_link_Class>
    <taxa_link_Family>1,4,5,17,29</taxa_link_Family>
    <taxa_link_Genus>22,50,57</taxa_link_Genus>
    <taxa_link_Order>1,4,5,12</taxa_link_Order>
    <taxa_link_Phylum>10,13,16</taxa_link_Phylum>
    <taxa_link_Species>5</taxa_link_Species>
    <_link_ParasiteSpecies>20</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T13:14:27.75+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:37:58.23+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>76</AnalysisID>
    <UUID>2a9e5cb4-46b1-4fdb-bb68-5107415678b8</UUID>
    <MetaAnalysisID>2018_4</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/mi.2017.20</DOI>
    <Title>Helminth-induced alterations of the gut microbiota exacerbate bacterial colitis</Title>
    <JournalID>13</JournalID>
    <Year>2018</Year>
    <Authors>Su C, Su L, Li Y, Long SR, Chang J, Zhang W, Walker WA, Xavier RX, Cherayil BJ, Shi HN</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study analyzed the fecal microbiota composition of Heligmosomoides polygyrus-infected and -uninfected BALB/C mice. Recipient mice of the gut microbiota from helminth-infected wide-type, but not STAT6-deficient, BALB/C donors displayed increased faecal pathogen shedding and exacerbation of Citrobacter-induced colitis compared to recipients of microbiota from control donors.</PredominantChangesReported>
    <taxa_link_Family>0</taxa_link_Family>
    <taxa_link_Genus>7,13,18,29,43,47,58</taxa_link_Genus>
    <taxa_link_Order>0</taxa_link_Order>
    <taxa_link_Phylum>3,9</taxa_link_Phylum>
    <taxa_link_Species>3</taxa_link_Species>
    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T13:17:51.76+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:41:46.82+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>77</AnalysisID>
    <UUID>5cad4c32-31b4-4bd8-b3f2-ab6b38942af7</UUID>
    <MetaAnalysisID>2018_5</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pntd.0006620</DOI>
    <Title>Dynamic changes in human-gut microbiome in relation to a placebo-controlled anthelminthic trial in Indonesia</Title>
    <JournalID>18</JournalID>
    <Year>2018</Year>
    <Authors>Martin I, Djuardi Y, Sartono E, Rosa BA, Supali T, Mitreva M, Houwing-Duistermaat JJ, Yazdanbakhsh M</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>7</DBArchiveID>
    <ProjectNumber>nematode.net/ Indonesia_Microbiome.htm</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated changes in the gut bacteria profiles of faecal samples from 150 human subjects enrolled in a double blind placebo controlled trial conducted in an area endemic for soil transmitted helminths in Indonesia. Either placebo or albendazole were given every three months for a period of one and a half years. Helminth infection was assessed before and at 3 months after the last treatment round. No differences were observed  in terms of relative abundance of gut microbial species between helminth-infected and uninfected subjects and at post-treatment, and no differences were detected in gut microbiome composition between albendazole- and placebo-recipients. In subjects who remained infected post-anthelmintic treatment,  a significant difference in microbiome composition between albendazole- and placebo-recipients was detected, and largely attributed to alterations in the abundance of Bacteroidetes. Albendazole was more effective against Ascaris lumbricoides and hookworms but not against Trichuris trichiura.</PredominantChangesReported>
    <taxa_link_Phylum>1,3</taxa_link_Phylum>
    <_link_ParasiteSpecies>18,19,9,22,3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T13:22:58.47+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:42:37.62+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>78</AnalysisID>
    <UUID>e4423a7b-1843-4e78-9ae4-44b03891ae8b</UUID>
    <MetaAnalysisID>2018_6</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.1038/s41598-018-33937-3</DOI>
    <Title>A comprehensive analysis of the faecal microbiome and metabolome of Strongyloides stercoralis infected volunteers from a non-endemic area</Title>
    <JournalID>22</JournalID>
    <Year>2018</Year>
    <Authors>Jenkins TP, Formenti F, Castro C, Piubelli C, Perandin F, Buonfrate D, Otranto D, Griffin JL, Krause L, Bisoffi Z, Cantacessi C</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>3</DBArchiveID>
    <ProjectNumber>10.17632/n86dtjvmbv.1)</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study explored the impact of natural mono-infections by the human parasite Strongyloides stercoralis on the faecal microbiota and metabolic profiles of a cohort of human volunteers from a non-endemic area of northern Italy (S+ = 20), pre- and post-anthelmintic treatment, and compared the findings with data obtained from a cohort of uninfected controls from the same geographical area (S- = 11). Samples from S+post-treatment subjects, displayed a significant decrease of bacteria belonging to the order Turicibacterales (genus Turicibacter) and an increase of Enterobacteriales (in particular associated to the genus Shigella) compared to corresponding samples from S+pre-treatment. Faecal metabolite analysis detected marked increases in the abundance of selected amino acids in S+ subjects, and of short chain fatty acids in S− subjects. </PredominantChangesReported>
    <taxa_link_Class>9,16</taxa_link_Class>
    <taxa_link_Family>2,24</taxa_link_Family>
    <taxa_link_Genus>16,35,66,88</taxa_link_Genus>
    <taxa_link_Order>14,16,21</taxa_link_Order>
    <taxa_link_Phylum>6,7</taxa_link_Phylum>
    <_link_ParasiteSpecies>22</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T13:28:27.09+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:46:57.31+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>79</AnalysisID>
    <UUID>b839ff5b-2e01-4fa2-bad3-bde1c4d7e1ce</UUID>
    <MetaAnalysisID>2018_7</MetaAnalysisID>
    <HostSpeciesID_Control>19</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.ijpara.2017.11.003 </DOI>
    <Title>The relationships between faecal egg counts and gut microbial composition in UK Thoroughbreds infected by cyathostomins</Title>
    <JournalID>11</JournalID>
    <Year>2018</Year>
    <Authors>Peachey LE, Molena RA, Jenkins TP, Di Cesare A, Traversa D, Hodgkinson JE, Cantacessi C </Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>3</DBArchiveID>
    <ProjectNumber>http://doi.org/10.17632/g7chkjrp8f.1</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised the composition of the equine gut commensal flora associated with the presence, in faecal samples, of low (Clow = 18) and high (Chigh = 18) numbers of eggs of cyathostomin parasites prior to and 2 and 14 days following treatment with ivermectin. Anthelmintic treatment in Chigh was associated with a significant reduction of the bacterial Phylum TM7 14 days post-ivermectin administration, as well as a transient expansion of Adlercreutzia spp. at 2 days post-treatment.</PredominantChangesReported>
    <taxa_link_Class>0</taxa_link_Class>
    <taxa_link_Family>12,23,33,34,36,39</taxa_link_Family>
    <taxa_link_Genus>83,87,110</taxa_link_Genus>
    <taxa_link_Order>9,18,23</taxa_link_Order>
    <taxa_link_Phylum>4</taxa_link_Phylum>
    <_link_ParasiteSpecies>23</_link_ParasiteSpecies>
    <_link_InfectedSpecies>19</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-20T13:36:38.09+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:44:47.99+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>80</AnalysisID>
    <UUID>cec3c02e-9028-427f-8acf-c9fb7a184098</UUID>
    <MetaAnalysisID>2017_3</MetaAnalysisID>
    <HostSpeciesID_Control>20</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1007/s11259-017-9698-5 </DOI>
    <Title>Microbial community and ovine host response varies with early and late stages of Haemonchus contortus infection </Title>
    <JournalID>25</JournalID>
    <Year>2017</Year>
    <Authors>El-Ashram S, Al Nasr I, Abouhaje F, El-Kemary M, Huang G, Dinçel G, Mehmood R, Hu M, Suo X </Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated changes in abomasal microbial profiles of lambs experimentally infected with Haemonchus contortus, at 7 and 50 days post-infection, and compared to uninfected controls.  </PredominantChangesReported>
    <taxa_link_Genus>84</taxa_link_Genus>
    <_link_ParasiteSpecies>16</_link_ParasiteSpecies>
    <_link_InfectedSpecies>20</_link_InfectedSpecies>
    <_link_SampleSite>16,4</_link_SampleSite>
    <_link_SampleType>4</_link_SampleType>
    <CreatedOn>2019-06-20T13:41:40.86+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T13:14:44.59+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>81</AnalysisID>
    <UUID>87a417df-cce9-4941-be3e-5ff8c1aaf7f0</UUID>
    <MetaAnalysisID>2015_8</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.pntd.0003861</DOI>
    <Title>Differences in the faecal microbiome in Schistosoma haematobium infected children vs. uninfected children</Title>
    <JournalID>18</JournalID>
    <Year>2015</Year>
    <Authors>Kay GL, Millard A, Sergeant MJ, Midzi N, Gwisai R, Mduluza T, Ivens A, Nausch N, Mutapi F, Pallen M</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised the faecal microbiota profiles of children from the Murewa district in Zimbabwe infected by Schistosoma haematobium, pre- and post-treatment with praziquantel. </PredominantChangesReported>
    <taxa_link_Genus>84</taxa_link_Genus>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T12:09:26.14+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T14:06:34.83+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>82</AnalysisID>
    <UUID>eb27df3b-807f-453c-b51e-5df363c04220</UUID>
    <MetaAnalysisID>2016_5</MetaAnalysisID>
    <HostSpeciesID_Control>12</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.bbi.2015.07.006</DOI>
    <Title>Got worms? Perinatal exposure to helminths prevents persistent immune sensitization and cognitive dysfunction induced by early-life infection</Title>
    <JournalID>4</JournalID>
    <Year>2016</Year>
    <Authors>Williamson LL, McKenney EA, Holzknecht ZE, Belliveau C, Rawls JF, Poulton S, Parker W, Bilbo SD</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>4</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>4</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>4</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the effect of Hymenolepis diminuta infection on the gut microbiome of newborn rats born from male and female Sprague–Dawley rats (infected six weeks prior to breeding) 21 days after birth. </PredominantChangesReported>
    <taxa_link_Class>3,6</taxa_link_Class>
    <_link_ParasiteSpecies>13</_link_ParasiteSpecies>
    <_link_InfectedSpecies>12</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <CreatedOn>2019-06-21T12:14:23.38+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T13:34:59.85+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>83</AnalysisID>
    <UUID>238b9b00-7112-4a7e-92b7-072fe759c999</UUID>
    <MetaAnalysisID>2018_8</MetaAnalysisID>
    <HostSpeciesID_Control>19</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.3389/fphys.2018.00272</DOI>
    <Title>Strongyle infection and gut microbiota: profiling of resistant and susceptible horses over a grazing season</Title>
    <JournalID>8</JournalID>
    <Year>2018</Year>
    <Authors>Clark A, Sallé G, Ballan V, Reigner F, Meynadier A, Cortet J, Koch C, Riou M, Blanchard A, Mach N</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJNA413884</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated differences in faecal microbial profiles between female Welsh ponies (10 susceptible (S) and 10 resistant (R) to parasite infections) during 5 months on pasture. Samples for microbiota sequencing and analysis were collected prior to turn-out, as well as at 24, 43, 92 and 132 days post-turnout.</PredominantChangesReported>
    <taxa_link_Family>18</taxa_link_Family>
    <taxa_link_Genus>2,12,22,30,31,78,87,92</taxa_link_Genus>
    <_link_ParasiteSpecies>24</_link_ParasiteSpecies>
    <_link_InfectedSpecies>19</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T12:20:20.1+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T11:49:13.96+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>84</AnalysisID>
    <UUID>1dd7e3ef-3a3f-49e1-9629-13961e375753</UUID>
    <MetaAnalysisID>2018_9</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.exppara.2018.08.002</DOI>
    <Title>Intestinal fluke Metagonimus yokogawai infection increases probiotic Lactobacillus in mouse cecum</Title>
    <JournalID>5</JournalID>
    <Year>2018</Year>
    <Authors>Kim JY, Kim EM, Yi M, Lee J, Lee S, Hwang Y, Yong D, Sohn WM, Yong TS</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>SRP154312</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated qualitative and quantitative changes in caecal content microbiota from ICR mice 4 weeks following infection with Metagonimus yokogawai, in comparison with uninfected control mice.</PredominantChangesReported>
    <taxa_link_Family>6,14,26,32,35,37,38</taxa_link_Family>
    <taxa_link_Phylum>2,9</taxa_link_Phylum>
    <taxa_link_Species>2,4,6,7</taxa_link_Species>
    <_link_ParasiteSpecies>25</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <CreatedOn>2019-06-21T12:29:07.1+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T11:40:39.33+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>85</AnalysisID>
    <UUID>07cca994-9ce4-4c19-bc7f-241623245e39</UUID>
    <MetaAnalysisID>2018_10</MetaAnalysisID>
    <HostSpeciesID_Control>4</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.3389/fimmu.2018.02557</DOI>
    <Title>Mucosal barrier and Th2 immune responses are enhanced by dietary inulin in pigs infected with Trichuris suis</Title>
    <JournalID>6</JournalID>
    <Year>2018</Year>
    <Authors>Myhill LJ, Stolzenbach S, Hansen TVA, Skovgaard K, Stensvold CR, O'Brien Andersen L, Nejsum P, Mejer H, Thamsborg SM, Williams AR</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJEB29079</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the effects of Trichuris suis infection on the gut microbiota of pigs supplemented with inulin. Inulin supplementation reduced the abundance of bacterial phyla linked to inflammation, such as Proteobacteria and Firmicutes, and simultaneously increased Actinobacteria and Bacteroidetes. Pigs treated with both inulin and T. suis displayed the highest Bacteroidetes: Firmicutes ratio and the lowest gut pH. </PredominantChangesReported>
    <taxa_link_Family>19</taxa_link_Family>
    <taxa_link_Genus>84</taxa_link_Genus>
    <taxa_link_Phylum>3,9</taxa_link_Phylum>
    <_link_ParasiteSpecies>4</_link_ParasiteSpecies>
    <_link_InfectedSpecies>4</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <CreatedOn>2019-06-21T12:51:56.15+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:50:10.51+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>86</AnalysisID>
    <UUID>7023ef7b-e64c-40ba-bde7-e8a629d4e74d</UUID>
    <MetaAnalysisID>2018_11</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.3389/fmicb.2018.02292</DOI>
    <Title>Altered gut microbiota composition in subjects infected with Clonorchis sinensis</Title>
    <JournalID>7</JournalID>
    <Year>2018</Year>
    <Authors>Xu M, Jiang Z, Huang W, Yin J, Ou S, Jiang Y, Meng L, Cao S, Yu A, Cao J, Shen Y</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>SRP158183</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study characterised qualitative and quantitative differences in faecal microbial profiles between 47 volunteers infected by Clonorchis sinensis and 42 healthy controls from five rural communities from the Teng County, Guangxi Zhuang Autonomous Region, China.</PredominantChangesReported>
    <taxa_link_Genus>6,13,17,23,27,28,29,38,39,40,41,42,44,57,62,64,67,68,73,80,81,89,92,97,100,111,112</taxa_link_Genus>
    <taxa_link_Phylum>3,5,15</taxa_link_Phylum>
    <taxa_link_Species>3</taxa_link_Species>
    <_link_ParasiteSpecies>26</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T14:25:03.16+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T12:39:13.29+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>87</AnalysisID>
    <UUID>da4b7dc2-d3e0-4192-9001-1a7c2b1ed415</UUID>
    <MetaAnalysisID>2018_12</MetaAnalysisID>
    <HostSpeciesID_Control>12</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1017/S0031182018000896</DOI>
    <Title>The benign helminth Hymenolepis diminuta ameliorates chemically induced colitis in a rat model system</Title>
    <JournalID>15</JournalID>
    <Year>2018</Year>
    <Authors>Pomajbíková KJ, Jirků M, Levá J, Sobotková K, Morien E, Parfrey LW</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJEB25354</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the ability of different life cycle stages of H. diminuta to protect rats against a model of colitis induced through application of the haptenizing agent dinitrobenzene sulphonic acid (DNBS) directly to the colon, partly via modifications in gut microbiota composition. The gut microbiota was disrupted during the onset of colitis and did not appear to play an overt role in H. diminuta-mediated protection.</PredominantChangesReported>
    <taxa_link_Family>41</taxa_link_Family>
    <taxa_link_Genus>7,45,58,60</taxa_link_Genus>
    <_link_ParasiteSpecies>13</_link_ParasiteSpecies>
    <_link_InfectedSpecies>12</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T14:38:52.36+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T12:35:42.09+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>88</AnalysisID>
    <UUID>802d5ed7-c7a6-48cc-9617-ac852bada887</UUID>
    <MetaAnalysisID>2018_13</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>DOI: 10.1126/sciadv.aap7399</DOI>
    <Title>Manipulation of host and parasite microbiotas: survival strategies during chronic nematode infection</Title>
    <JournalID>21</JournalID>
    <Year>2018</Year>
    <Authors>White EC, Houlden A, Bancroft AJ, Hayes KS, Goldrick M, Grencis RK, Roberts IS</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJEB12611</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated the impact of Trichuris muris–induced alterations in the host intestinal microbiota on the outcome of subsequent rounds of infection. The study demonstrated that a second infection by Trichuris muris resulted, therefore hatching in an altered host intestinal microbiota, resulted in significantly reduced worm burden compared to those infecting a naïve host intestinal microbiota.</PredominantChangesReported>
    <taxa_link_Family>33,35</taxa_link_Family>
    <taxa_link_Phylum>3,9</taxa_link_Phylum>
    <_link_ParasiteSpecies>10</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <CreatedOn>2019-06-21T14:43:15.24+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T12:32:31.63+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>89</AnalysisID>
    <UUID>20ea7ab7-3969-4e86-b680-c20a72b1f9c2</UUID>
    <MetaAnalysisID>2019_1</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.7717/peerj.6200</DOI>
    <Title>Intestinal parasitic infection alters bacterial gut microbiota in children</Title>
    <JournalID>16</JournalID>
    <Year>2019</Year>
    <Authors>Toro-Londono MA, Bedoya-Urrego K, Garcia-Montoya GM, Galvan-Diaz AL, Alzate JF​</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA487588</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated qualitative and quantitative differences in faecal microbiota composition between children infected by Cryptosporidium (n = 5), Giardia intestinalis (n = 6), and co-infected by Ascaris sp. and either Trichuris trichiura or Enterobius vermicularis or Giardia (n = 6). Samples from uninfected controls (n = 6) were also included. The latter group was characterised a type I enterotype (i.e. predominant Bacteroides spp.), whilst helminth- and protozoan-infected children are characterised by a type II enterotype (predominant Prevotella spp.).  </PredominantChangesReported>
    <taxa_link_Family>2,33</taxa_link_Family>
    <taxa_link_Genus>13,84</taxa_link_Genus>
    <_link_ParasiteSpecies>19,143,39,30,31,3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T14:47:07.33+00:00</CreatedOn>
    <UpdatedOn>2019-06-25T10:35:15.06+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>90</AnalysisID>
    <UUID>9cc322a8-4b66-4ddd-ada9-3165e81f5879</UUID>
    <MetaAnalysisID>2019_2</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/s41598-018-36709-1</DOI>
    <Title>Urogenital schistosomiasis is associated with signatures of microbiome dysbiosis in Nigerian adolescents</Title>
    <JournalID>22</JournalID>
    <Year>2019</Year>
    <Authors>Ajibola O, Rowan AD, Ogedengbe CO, Mshelia MB, Cabral DJ, Eze AA, Obaro S, Belenky P</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>1</DBArchiveID>
    <ProjectNumber>https://repository.library.brown.edu/studio/item/bdr:698310/</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated qualitative and quantitative differences in faecal microbiota composition between Schistosoma haematobium-infected (n = 24) and -uninfected (n = 25) young adolescents from the Argungu Local Government Area of Kebbi State, Nigeria. </PredominantChangesReported>
    <taxa_link_Class>1,5,6,8,13,15,17,18</taxa_link_Class>
    <taxa_link_Family>3,10,14,25,28,29,33,36,40</taxa_link_Family>
    <taxa_link_Genus>3,9,29,34,37,38,53,64,69,74,80,83,84,102</taxa_link_Genus>
    <taxa_link_Order>5,7,12,13,17,18,20,22</taxa_link_Order>
    <taxa_link_Phylum>4,9,13,15</taxa_link_Phylum>
    <_link_ParasiteSpecies>27</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T14:51:36.08+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T12:56:57.37+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>91</AnalysisID>
    <UUID>b43c8f44-e4ab-40b0-8fcc-51c996985361</UUID>
    <MetaAnalysisID>2019_3</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1007/s00436-018-6179-x</DOI>
    <Title>Chinese liver fluke Clonorchis sinensis infection changes the gut microbiome and increases probiotic Lactobacillus in mice</Title>
    <JournalID>14</JournalID>
    <Year>2019</Year>
    <Authors>Kim JY, Kim EM, Yi M, Lee J, Lee S, Hwang Y, Yong D, Sohn WM, Yong TS</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>Unavailable</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated changes in the gut microbiome of C57BL/6 mice infected with Clonorchis sinensis metacercariae over time. Differences in gut microbiome composition between infected and uninfected mice were detected at 20 and 30 days post-infection, whereas no significant differences between groups were observed at 50 days post-infection.</PredominantChangesReported>
    <taxa_link_Genus>58</taxa_link_Genus>
    <taxa_link_Species>7</taxa_link_Species>
    <_link_ParasiteSpecies>26</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-06-21T15:02:29.58+00:00</CreatedOn>
    <UpdatedOn>2019-07-04T10:27:01.97+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>92</AnalysisID>
    <UUID>0a414510-e7d8-4e9e-8e54-aa7a805d3652</UUID>
    <MetaAnalysisID>2015_8</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.ijpara.2015.04.001</DOI>
    <Title>(CAT) Differences in the faecal microbiome of non-diarrhoeic clinically healthy dogs and cats associated with Giardia duodenalis infection: impact of hookworms and coccidia</Title>
    <JournalID>11</JournalID>
    <Year>2015</Year>
    <Authors>Šlapeta J, Dowd SE, Alanazi AD, Westman ME, Brown GK</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>6</DBArchiveID>
    <ProjectNumber>PRJNA276586</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study compared the composition and diversity of bacterial communities from 40 dogs, including free-roaming dogs, and 21 surrendered cats from Australia. The dog cohort included 17 (42.5%) dogs positive for Giardia and 13 (32.5%) dogs positive for dog hookworm (Ancylostoma caninum). The cat samples included eight positive for Giardia and eight positive for Cystoisospora. In dogs, significant difference in faecal microbial profiles (at genus level) were observed between the Giardia-positive and -negative groups. No such difference was demonstrated between Ancylostoma-positive and -negative dogs. </PredominantChangesReported>
    <taxa_link_Genus>13,18,24,29,51,55,86,87</taxa_link_Genus>
    <_link_ParasiteSpecies>142,31</_link_ParasiteSpecies>
    <_link_InfectedSpecies>24,13</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-07-04T15:16:38.49+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>93</AnalysisID>
    <UUID>a39e1599-b817-4384-83f3-2ee9d48c84e5</UUID>
    <MetaAnalysisID>2019_4</MetaAnalysisID>
    <HostSpeciesID_Control>20</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/s41396-019-0462-4</DOI>
    <Title>Microbiome analysis as a platform R&amp;D tool for parasitic nematode disease management</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Glenn Hogan, Sidney Walker, Frank Turnbull, Tania Curiao, Alison A. Morrison, Yensi Flores, Leigh Andrews, Marcus J. Claesson, Mark Tangney, Dave J. Bartley </Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated, pre-and post-infection, qualitative and quantitative changes in abomasum and feaces content microbiota of sheep co-infected with Haemonchus contortus and Teladorsagia circumcincta.
</PredominantChangesReported>
    <_link_ParasiteSpecies>16</_link_ParasiteSpecies>
    <_link_InfectedSpecies>20</_link_InfectedSpecies>
    <_link_SampleSite>16</_link_SampleSite>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-07-28T23:20:07.91+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>94</AnalysisID>
    <UUID>3f91ce99-938f-4e57-839f-8256ef3b0ae5</UUID>
    <MetaAnalysisID>2019_5</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.3390/cells8060577</DOI>
    <Title>Contribution of the gut microbiota in P28GST-mediated anti-inflammatory effects: experimental and clinical insights</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Benoît Foligné , Coline Plé, Marie Titécat, Arnaud Dendooven, Aurélien Pagny, Catherine Daniel, Elisabeth Singer, Muriel Pottier, Benjamin Bertin, Christel Neut, Dominique Deplanque, Laurent Dubuquoy, Pierre Desreumaux, Monique Capron, Annie Standaert</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigatd the changes in the profiles of mice faecal microbiota at several time points of the P28GST-immunomodulation period.
</PredominantChangesReported>
    <_link_ParasiteSpecies>145</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <CreatedOn>2019-07-28T23:21:35.53+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>95</AnalysisID>
    <UUID>8dc1fa22-3a00-4c68-9aaf-409b87821f55</UUID>
    <MetaAnalysisID>2019_6</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>4</ProfilingMethodID>
    <DOI>https://doi.org/10.1128/IAI.00275-19</DOI>
    <Title>Schistosoma mansoni worm infection regulates the intestinal microbiota and susceptibility to colitis</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Achilleas Floudasa, Gabriella Avielloa, Christian Schwartza, Ian B. Jefferyb, Paul W. O'Tooleb and Padraic G. Fallon</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>5</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>4</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>4</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>4</BetaDiversity_ChanageId>
    <PredominantChangesReported>The study investigated the faecal microbiota profile of mice infected or with only adult male Schistosoma mansoni worms, or co-infected with adult male- and female-worms. 
</PredominantChangesReported>
    <_link_ParasiteSpecies>20</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <CreatedOn>2019-07-28T23:23:21.9+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>96</AnalysisID>
    <UUID>3c55975e-fdd4-4377-b2bb-67905b0f87e0</UUID>
    <MetaAnalysisID>2019_7</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1128/mBio.00519-19
</DOI>
    <Title>The impact of anthelmintic treatment on human gut microbiota based on cross-sectional and pre- and postdeworming comparisons in Western Kenya</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Alice V. Easton, Mariam Quiñones, Ivan Vujkovic-Cvijin, Rita G. Oliveira, Stella Kepha, Maurice R. Odiere, Roy M. Anderson, Yasmine Belkaid, Thomas B. Nutman</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study analysed the microbiota profiles of stool samples collected from residents of 5 rural Kenyan villages prior to and 3 weeks and 3 months following albendazole therapy, to provide new insight into the complex interplay among soil-transmitted helminths and human gut microbiome. 
</PredominantChangesReported>
    <_link_ParasiteSpecies>19,9</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <CreatedOn>2019-07-28T23:24:05.14+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>97</AnalysisID>
    <UUID>a2a7077f-95cd-4012-8282-929548e0ffd7</UUID>
    <MetaAnalysisID>2019_8</MetaAnalysisID>
    <HostSpeciesID_Control>19</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1016/j.ijpara.2019.02.003
</DOI>
    <Title>Removal of adult cyathostomins alters faecal microbiota and promotes an inflammatory phenotype in horses</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Nicola Walshe, Vivienne Duggan, Raul Cabrera-Rubio, Fiona Crispie, Paul Cotter, Orna Feehan, Grace Mulcahy</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>4</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, authors studied changes in the faecal microbiota of two groups of horses, infected by cyathostomins,  following treatment with anthelmintics (fenbendazole or moxidectin). 
</PredominantChangesReported>
    <_link_ParasiteSpecies>23</_link_ParasiteSpecies>
    <_link_InfectedSpecies>19</_link_InfectedSpecies>
    <CreatedOn>2019-07-28T23:24:54.72+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>98</AnalysisID>
    <UUID>a6d40ad3-55f0-4f9a-92ee-507b1cd0b76d</UUID>
    <MetaAnalysisID>2019_9</MetaAnalysisID>
    <HostSpeciesID_Control>5</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.4269/ajtmh.18-0968</DOI>
    <Title>Interactions between parasitic infections and the human gut microbiome in Odisha, India
</Title>
    <JournalID>29</JournalID>
    <Year>2019</Year>
    <Authors>Tiffany Huwe, Birendra Kumar Prusty, Aisurya Ray, Shaun Lee, Balachandran Ravindran, Edwin Michael</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>0</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, authors investigated the human faecal microbiota profile associated with soil-transmitted helminth (Ascaris lumbricoides, Necator americanus, Trichuris trichiura) infections in the area of Odisha, India. </PredominantChangesReported>
    <_link_ParasiteSpecies>19,9,3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleSite>0</_link_SampleSite>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-08-05T10:59:19.32+00:00</CreatedOn>
    <UpdatedOn>2019-08-05T11:35:13.27+00:00</UpdatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>99</AnalysisID>
    <UUID>d0c95d03-7d0b-45ca-b4c9-93d374d291da</UUID>
    <MetaAnalysisID>2019_10</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1128/IAI.00042-19</DOI>
    <Title>Suppression of obesity by an intestinal helminth through interactions with intestinal microbiota
</Title>
    <JournalID>1</JournalID>
    <Year>2019</Year>
    <Authors>Chikako Shimokawa, Seiji Obi, Mioko Shibata, Alex Olia, Takashi Imai, Kazutomo Suzue, Hajime Hisaeda</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>New project number</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>4</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>4</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>4</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigated whether an intestinal nematode, Heligmosomoides polygyrus, has a suppressive role in diet-induced obesity in mice, altering the intestinal microbiota profile.
</PredominantChangesReported>
    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>0</_link_SampleSite>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-08-05T11:06:07.34+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>100</AnalysisID>
    <UUID>ca75498e-2c75-44ad-b880-3ed20a37ac6c</UUID>
    <MetaAnalysisID>2019_11</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>256</ProfilingMethodID>
    <DOI>https://doi.org/10.1038/s41467-019-09361-0</DOI>
    <Title>The parasitic worm product ES-62 normalises the gut microbiota bone marrow axis in inflammatory arthritis</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>James Doonan, Anuradha Tarafdar, Miguel A. Pineda, Felicity E. Lumb, Jenny Crowe, Aneesah M. Khan, Paul A. Hoskisson, Margaret M. Harnett, William Harnett </Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>mgm4777616.3, 4777615.3, 4777614.3, 4777613.3, 4777481.3, 4777480.3, 4777479.3, 4777478.3, 4767994.3, 4767993.3, 4767992.3, 4767991.3, 4767990.3, 4767989.3, 4767988.3, 4767987.3, 4767986.3, 4738191.3, 4738190.3, 4738025.3, 4737887.3, 4737053.3 and 4737052.3</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>1</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>1</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>4</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, authors employ ES-62, an immunomodulator secreted by tissue-dwelling Acanthocheilonema viteae to show that helminth-modulation of the gut microbiome does not require live infection with gastrointestinal-based worms. 
</PredominantChangesReported>
    <_link_ParasiteSpecies>147,148</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <CreatedOn>2019-08-05T11:07:38.39+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>101</AnalysisID>
    <UUID>b5a6798d-572b-4337-91b6-fe6800e3a043</UUID>
    <MetaAnalysisID>2019_12</MetaAnalysisID>
    <HostSpeciesID_Control>1</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.1371/journal.ppat.1007265</DOI>
    <Title>Exclusive dependence of IL-10Rα signalling on intestinal microbiota homeostasis and control of whipworm infection</Title>
    <JournalID>19</JournalID>
    <Year>2019</Year>
    <Authors>María A. Duque-Correa , Natasha A. Karp, Catherine McCarthy, Simon Forman, David Goulding, Geetha Sankaranarayanan, Timothy P. Jenkins, Adam J. Reid, Emma L. Cambridge, Carmen Ballesteros Reviriego, The Sanger Mouse Genetics Project, The 3i consortium , Werner Müller, Cinzia Cantacessi, Gordon Dougan, Richard K. Grencis, Matthew Berriman</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>ERP016361</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>2</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>2</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>1</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study analysed the role of the IL-10 family, an intestinal inflammatory responses mediators group, in the resistance of mice to infection with Trichuris muris, investigating the caecal microbiota profile.  
</PredominantChangesReported>
    <_link_ParasiteSpecies>10</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleSite>2</_link_SampleSite>
    <CreatedOn>2019-08-05T11:09:57.87+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>102</AnalysisID>
    <UUID>9ff2fb8b-2a3c-4cd9-9dd0-7b9e308d83c1</UUID>
    <MetaAnalysisID>2019_13</MetaAnalysisID>
    <HostSpeciesID_Control>19</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>In press</DOI>
    <Title>Dysbiosis associated with acute helminth infections in herbivorous youngstock – observations and implications</Title>
    <JournalID>22</JournalID>
    <Year>2019</Year>
    <Authors>Laura E. Peachey, Cecilia Castro, Rebecca A. Molena, Timothy P. Jenkins, Julian L. Griffin, Cinzia Cantacessi</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>3</DBArchiveID>
    <ProjectNumber>DOI: 10.17632/95m8sfd3kt.1</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>This study investigates, for the first time, the associations between acute infections by gastrointestinal helminths and the faecal microbial of a cohort of equine youngstock, prior to and following treatment with parasiticides (ivermectin). 
</PredominantChangesReported>
    <_link_ParasiteSpecies>146,24</_link_ParasiteSpecies>
    <_link_InfectedSpecies>19</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2019-08-05T11:26:28.77+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>103</AnalysisID>
    <UUID>d781f0f1-2a07-45e5-96f8-64b3ab2be2aa</UUID>
    <MetaAnalysisID>2019_14</MetaAnalysisID>
    <HostSpeciesID_Control>4</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>https://doi.org/10.3389/fimmu.2018.02557</DOI>
    <Title>Mucosal barrier and Th2 immune responses are enhanced by dietary inulin in pigs infected with Trichuris suis</Title>
    <JournalID>6</JournalID>
    <Year>2019</Year>
    <Authors>Laura J. Myhill, Sophie Stolzenbach, Tina V. A. Hansen, Kerstin Skovgaard, C. Rune Stensvold, Lee O'Brien Andersen, Peter Nejsum, Helena Mejer, Stig M. Thamsborg, Andrew R. Williams</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJEB29079</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, authors investigated the colon microbiota profile of pigs infected with the whipworm Trichuris suis and feeded with inulin.
</PredominantChangesReported>
    <_link_ParasiteSpecies>4</_link_ParasiteSpecies>
    <_link_InfectedSpecies>4</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <CreatedOn>2019-08-05T11:28:11.36+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>104</AnalysisID>
    <UUID>b82fe7e3-7e91-43ad-869d-b6d24b158ec6</UUID>
    <MetaAnalysisID>2019_15</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.3389/fcimb.2019.00217</DOI>
    <Title>Sequential Changes in the Host Gut Microbiota During Infection With the Intestinal Parasitic Nematode Strongyloides venezuelensis</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Tanzila Afrin, Kazunori Murase, Asuka Kounosu, Vicky L. Hunt, Mark Bligh, Yasunobu Maeda, Akina Hino, Haruhiko Maruyama, Isheng J. Tsai, and Taisei Kikuchi</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>SAMD00148911</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>TODO 2020-04-25</PredominantChangesReported>
    <_link_ParasiteSpecies>24</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2020-05-24T15:33:57.04+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>105</AnalysisID>
    <UUID>2a889f5c-dfb2-4e84-9410-72700b9cba3a</UUID>
    <MetaAnalysisID>2019_16</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.1038/s41598-019-44301-4</DOI>
    <Title>Cognitive and Microbiome Impacts of Experimental Ancylostoma ceylanicum Hookworm Infections in Hamsters</Title>
    <JournalID>22</JournalID>
    <Year>2019</Year>
    <Authors>Samuel C. Pan, Doyle V. Ward, Yunqiang Yin, Yan Hu, Mostafa A. Elfawal, Robert E. Clark, and Raffi V. Aroian</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>PRJNA535443</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>Here, authors studied the impact of a human hookworm parasite, Ancylostoma ceylanicum, in hamster gut microbiome
</PredominantChangesReported>
    <_link_ParasiteSpecies>0</_link_ParasiteSpecies>
    <_link_InfectedSpecies>7</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2020-05-24T15:38:08.09+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>106</AnalysisID>
    <UUID>ccb10f63-dce8-4001-809b-2f1a19645940</UUID>
    <MetaAnalysisID>2019_17</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.1016/j.ijpara.2018.10.007</DOI>
    <Title>Ascaris suum infection was associated with a worm-independent reduction in microbial diversity and altered metabolic potential in the porcine gut microbiome.</Title>
    <JournalID>11</JournalID>
    <Year>2019</Year>
    <Authors>Yueying Wang, Fang Liu, Joseph F. UrbanJr., Oonagh Paerewijck, Peter Geldhof, Robert W.Li</Authors>
    <IsOpenAccess>false</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>SRP127199</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>In the present study, authors analized the effect of infection of Ascaris suum on the microbial composition in pigs
</PredominantChangesReported>
    <_link_ParasiteSpecies>0</_link_ParasiteSpecies>
    <_link_InfectedSpecies>4</_link_InfectedSpecies>
    <_link_SampleSite>128</_link_SampleSite>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2020-05-24T16:05:40.39+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>107</AnalysisID>
    <UUID>9ec071f6-b707-45e4-845a-e44bac977771</UUID>
    <MetaAnalysisID>2019_18</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>64</ProfilingMethodID>
    <DOI>10.3389/fgene.2019.01028</DOI>
    <Title>The Effect of Gut Microbiome Composition on Human Immune Responses: An Exploration of Interference by Helminth Infections</Title>
    <JournalID>0</JournalID>
    <Year>2019</Year>
    <Authors>Ivonne Martin, Maria M. M. Kaisar,  Aprilianto E. Wiria,Firdaus Hamid, Yenny Djuardi, Erliyani Sartono, Bruce A. Rosa,  Makedonka Mitreva, Taniawati Supali, Jeanine J. Houwing-Duistermaat,  Maria Yazdanbakhsh, and Linda J. Wammes</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>0</DBArchiveID>
    <ProjectNumber>SAMN07688522 to SAMN07688545</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study authors aim to analyze the relationship between bacterial communities with cytokine response in the presence or absence of helminth infections.
gcdsdbchj fr</PredominantChangesReported>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2020-05-24T16:14:27.27+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>108</AnalysisID>
    <UUID>30d238fd-10c7-4a98-ab5b-3f9b2e449b2a</UUID>
    <MetaAnalysisID>2019_19</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>2</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.1371/journal.ppat.1008066</DOI>
    <Title>Linking the effects of helminth infection, diet and the gut microbiota with human whole-blood signatures</Title>
    <JournalID>19</JournalID>
    <Year>2019</Year>
    <Authors>Soo Ching Lee, Mei San Tang, Alice V. Easton, Joseph Cooper Devlin, Ling Ling Chua, Ilseung Cho, Foong Ming Moy, Tsung Fei Khang, Yvonne A. L. Lim , P’ng Loke</Authors>
    <IsOpenAccess>true</IsOpenAccess>
    <DBArchiveID>2</DBArchiveID>
    <ProjectNumber>PRJEB34956 and PRJEB34957</ProjectNumber>
    <AlphaDiversity_EvennessChanageId>0</AlphaDiversity_EvennessChanageId>
    <AlphaDiversity_RichnessChanageId>0</AlphaDiversity_RichnessChanageId>
    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>Here authors analyzed the relationship between fecal microbiota and Trichuris trichiura infection inindigenous Malaysians, referred to locally as Orang Asli, and urban participants from the capital city of Malaysia, Kuala Lumpur
</PredominantChangesReported>
    <_link_ParasiteSpecies>3</_link_ParasiteSpecies>
    <_link_InfectedSpecies>5</_link_InfectedSpecies>
    <_link_SampleType>2</_link_SampleType>
    <CreatedOn>2020-05-24T16:26:32.5+00:00</CreatedOn>
    <Active>true</Active>
  </Microbiota.Analysis>
  <Microbiota.Analysis>
    <AnalysisID>109</AnalysisID>
    <UUID>22ae3c60-7388-42bf-a87c-e4168520f980</UUID>
    <MetaAnalysisID>2019_20</MetaAnalysisID>
    <HostSpeciesID_Control>0</HostSpeciesID_Control>
    <InfectionID>1</InfectionID>
    <ProfilingMethodID>8</ProfilingMethodID>
    <DOI>10.1016/j.jmii.2019.09.009</DOI>
    <Title>Trichinella spiralis infection decreases the diversity of the intestinal flora in the infected mouse.</Title>
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    <Year>2019</Year>
    <Authors>Sha Liu, Jin Pan, Xiangli Meng, Junping Zhu, Jie Zhou, Xinping Zhu</Authors>
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    <DOI>10.1016/j.meegid.2019.104010.</DOI>
    <Title>Exploring interactions between Blastocystis sp., Strongyloides spp. and the gut microbiomes of wild chimpanzees in Senegal</Title>
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    <Authors>Justinn Renelies-Hamilton, Marc Noguera-Julian, Mariona Parera, Roger Paredes, Liliana Pacheco,  Elena Dacal, José M.Saugar, José M.Rubio, Michael Poulsen, Pamela C.Köster, David Carmena</Authors>
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    <PredominantChangesReported>Authors studied the interaction between gut microbiome and enteric parasites in chimpanzees.</PredominantChangesReported>
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    <DOI>10.1016/j.vetmic.2020.108607</DOI>
    <Title>Gut microbial signatures associated with moxidectin treatment efficacy of Haemonchus contortus in infected goats.</Title>
    <JournalID>0</JournalID>
    <Year>2020</Year>
    <Authors>Fang Liu, Yue Xie, Anne M.Zajac, Yan Hu, Raffi V.Aroian, Joseph F.Urban Jr., Robert W.Li</Authors>
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    <DOI>10.1016/j.ijpara.2019.09.005</DOI>
    <Title>Infection with a small intestinal helminth, Heligmosomoides polygyrus bakeri, consistently alters microbial communities throughout the murine small and large intestine.</Title>
    <JournalID>11</JournalID>
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    <Authors>Alexis Rapin, Audrey Chuat, Luc Lebon, Mario M. Zaiss, Benjamin J. Marsland, Nicola L. Harris</Authors>
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    <_link_ParasiteSpecies>1</_link_ParasiteSpecies>
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    <InfectionID>1</InfectionID>
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    <DOI>10.1038/s41396-020-0589-3</DOI>
    <Title>The gut microbiota response to helminth infection depends on host sex and genotype.</Title>
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    <Year>2020</Year>
    <Authors>Fei Ling, Natalie Steinel, Jesse Weber, Lei Ma, Chris Smith, Decio Correa, Bin Zhu, Daniel Bolnick &amp; Gaoxue Wang </Authors>
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    <InfectionID>1</InfectionID>
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    <DOI>10.1080/19490976.2019.1688065</DOI>
    <Title>Worm expulsion is independent of alterations in composition of the colonic bacteria that occur during experimental Hymenolepis diminuta-infection in mice.</Title>
    <JournalID>9</JournalID>
    <Year>2020</Year>
    <Authors>Adam Shute, Arthur Wang, Timothy S. Jayme, Marc Strous, Kathy D. McCoy, Andre G. Buret, Derek M. McKay</Authors>
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    <PredominantChangesReported>In this study, authors analized the interaction between the tapeworm Hymenolepis diminuta and the gut microbiome in mice.
</PredominantChangesReported>
    <_link_ParasiteSpecies>13</_link_ParasiteSpecies>
    <_link_InfectedSpecies>1</_link_InfectedSpecies>
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    <CreatedOn>2020-05-24T19:34:02.73+00:00</CreatedOn>
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    <DOI>10.1186/s40168-020-00818-9</DOI>
    <Title>Infection with the sheep gastrointestinal nematode Teladorsagia circumcincta increases luminal pathobionts</Title>
    <JournalID>0</JournalID>
    <Year>2020</Year>
    <Authors>Alba Cortés, John Wills, Xiaopei Su, Rachel E. Hewitt, Jack Robertson, Riccardo Scotti, Daniel R. G. Price, Yvonne Bartley, Tom N. McNeilly, Lutz Krause, Jonathan J. Powell, Alasdair J. Nisbet, Cinzia Cantacessi</Authors>
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    <BetaDiversity_ChanageId>0</BetaDiversity_ChanageId>
    <PredominantChangesReported>In this study, authors investigated the fluctuations in microbiota composition of sheep vaccinated against, and experimentally infected with, the ‘brown stomach worm’ Teladorsagia circumcincta.

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    <InfectionID>1</InfectionID>
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    <DOI>10.21203/rs.2.24428/v1</DOI>
    <Title>Experimental infection with the hookworm, Necator americanus, promotes gut microbial diversity in human volunteers with relapsing multiple sclerosis</Title>
    <JournalID>0</JournalID>
    <Year>2020</Year>
    <Authors>Timothy Jenkins, David Pritchard, Radu Tanasescu, Gary Telford, Marina Paraiakovou, Cris Constantinescu, Cinzia Cantacessi</Authors>
    <IsOpenAccess>true</IsOpenAccess>
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    <PredominantChangesReported>In the present study authors investigated qualitative and quantitative changes in gut microbial composition of human volunteers with remitting multiple sclerosis (RMS) prior to and following experimental infection with the human hookworm, Necator americanus, and following anthelmintic treatment, and compared the findings with data obtained from a cohort of RMS patients subjected to placebo treatment (PBO)
</PredominantChangesReported>
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    <CreatedOn>2020-05-24T19:46:02.09+00:00</CreatedOn>
    <Active>true</Active>
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    <Name>Metagonimus yokogawai</Name>
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    <Name>Schistosoma haematobium</Name>
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    <Name>Entamoeba hartmanni</Name>
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    <Name>Ancylostoma caninum</Name>
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    <ParasiteSpeciesID>144</ParasiteSpeciesID>
    <Name>Teladorsagia circumcincta</Name>
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    <ParasiteSpeciesID>145</ParasiteSpeciesID>
    <Name>glutathione S-transferase (P28GST) from Schistosoma
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    <ParasiteSpeciesID>146</ParasiteSpeciesID>
    <Name>Anoplocephala perfoliata</Name>
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    <Name>ES-62</Name>
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    <Name>glycoprotein from Acanthocheilonema viteae</Name>
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    <Name>Trichinella spiralis
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    <Name>Blastocystis sp.</Name>
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    <Name>Paraprevotellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7968b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>28</FamilyId>
    <Name>Pasteurellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7a68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>29</FamilyId>
    <Name>Peptococcaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7b68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>30</FamilyId>
    <Name>Peptostreptococcaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7c68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>31</FamilyId>
    <Name>Phyromonadaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7d68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>32</FamilyId>
    <Name>Porphyromonadaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7e68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>33</FamilyId>
    <Name>Prevotellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>7f68b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>34</FamilyId>
    <Name>Pseudomonadaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8068b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>35</FamilyId>
    <Name>Rikenellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8168b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>36</FamilyId>
    <Name>Ruminococcaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8268b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>37</FamilyId>
    <Name>S24-7</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8368b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>38</FamilyId>
    <Name>Sutterellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8468b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>39</FamilyId>
    <Name>Turicibacteraceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8568b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>40</FamilyId>
    <Name>Veillonellaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8668b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>41</FamilyId>
    <Name>Verrucomicrobiaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8768b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>42</FamilyId>
    <Name>Weeksellacea</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8868b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Family>
    <FamilyId>43</FamilyId>
    <Name>Lachnospiracaea</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>3af71061-46f2-e911-a40b-281878345184</UUID>
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  <Taxonomy.Family>
    <FamilyId>44</FamilyId>
    <Name>Mogibacteriaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>df6eb5ed-48f2-e911-a40b-281878345184</UUID>
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  <Taxonomy.Family>
    <FamilyId>45</FamilyId>
    <Name>Dehalobacteriaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e06eb5ed-48f2-e911-a40b-281878345184</UUID>
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  <Taxonomy.Family>
    <FamilyId>46</FamilyId>
    <Name>Streptococcaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e16eb5ed-48f2-e911-a40b-281878345184</UUID>
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  <Taxonomy.Genus>
    <GenusId>0</GenusId>
    <Name>-- N/A --</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8968b104-9c99-e911-abc4-000d3a228e9c</UUID>
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  <Taxonomy.Genus>
    <GenusId>1</GenusId>
    <Name>Acetanaerobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8a68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>2</GenusId>
    <Name>Acetivibrio</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8b68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>3</GenusId>
    <Name>Acinetobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8c68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>4</GenusId>
    <Name>Actinobacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8d68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>5</GenusId>
    <Name>Adlercreutzia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8e68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>6</GenusId>
    <Name>Agrobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>8f68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>7</GenusId>
    <Name>Akkermansia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>8</GenusId>
    <Name>Allobaculum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>9</GenusId>
    <Name>Alloprevotella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>10</GenusId>
    <Name>Aquamicrobium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>11</GenusId>
    <Name>Asteroleplasma</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>12</GenusId>
    <Name>Bacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>13</GenusId>
    <Name>Bacteroides</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>14</GenusId>
    <Name>Bacteroidescaecum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>15</GenusId>
    <Name>Barnsiella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>16</GenusId>
    <Name>BF311</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>17</GenusId>
    <Name>Bifidobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9a68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>18</GenusId>
    <Name>Blautia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9b68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>19</GenusId>
    <Name>Brachyspira</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9c68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>20</GenusId>
    <Name>Bulleidia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9d68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>21</GenusId>
    <Name>Butyricicoccus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9e68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>22</GenusId>
    <Name>Campylobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9f68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>23</GenusId>
    <Name>Candidatus Arthromitus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>24</GenusId>
    <Name>Catenibacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>25</GenusId>
    <Name>CF231</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>26</GenusId>
    <Name>Chelatococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>27</GenusId>
    <Name>Christensenella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>28</GenusId>
    <Name>Cloacibacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>29</GenusId>
    <Name>Clostridium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>30</GenusId>
    <Name>ClostridiumIII</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>31</GenusId>
    <Name>ClsotridiumXIVa</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>32</GenusId>
    <Name>Collinsella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>33</GenusId>
    <Name>Coprobacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>aa68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>34</GenusId>
    <Name>Coriobacteriaceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ab68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>35</GenusId>
    <Name>Dehalobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ac68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>36</GenusId>
    <Name>Desulfocella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ad68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>37</GenusId>
    <Name>Desulfovibrio</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ae68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>38</GenusId>
    <Name>Dialister</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>af68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>39</GenusId>
    <Name>Dorea</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>40</GenusId>
    <Name>Enhydrobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>41</GenusId>
    <Name>Enterobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>42</GenusId>
    <Name>Enterococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>43</GenusId>
    <Name>Enterorhabdus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>44</GenusId>
    <Name>Epulopiscium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>45</GenusId>
    <Name>Escherichia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>46</GenusId>
    <Name>Ethanoligenens</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>47</GenusId>
    <Name>Eubacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>48</GenusId>
    <Name>Fibrobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>b968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>49</GenusId>
    <Name>Francisella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ba68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>50</GenusId>
    <Name>Fructobacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>bb68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>51</GenusId>
    <Name>Fusobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>bc68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>52</GenusId>
    <Name>Geobacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>bd68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>53</GenusId>
    <Name>Haemophilus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>be68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>54</GenusId>
    <Name>Halomonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>bf68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>55</GenusId>
    <Name>Howardella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>56</GenusId>
    <Name>Jeotgalicoccus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>57</GenusId>
    <Name>Klebsiella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>58</GenusId>
    <Name>Lactobacillus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>59</GenusId>
    <Name>Lactobacillusileum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>60</GenusId>
    <Name>Lactococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>61</GenusId>
    <Name>Leptomena</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>62</GenusId>
    <Name>Leucobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>63</GenusId>
    <Name>Megamonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>64</GenusId>
    <Name>Megasphaera</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>c968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>65</GenusId>
    <Name>Meiothermus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ca68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>66</GenusId>
    <Name>Methanocorpusculum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>cb68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>67</GenusId>
    <Name>Methylobacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>cc68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>68</GenusId>
    <Name>Microbacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>cd68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>69</GenusId>
    <Name>MollicutesRF9</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ce68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>70</GenusId>
    <Name>Mucispirillum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>cf68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>71</GenusId>
    <Name>Mucospirillium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>72</GenusId>
    <Name>Mycoplasma</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>73</GenusId>
    <Name>Nevskia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>74</GenusId>
    <Name>Olsenella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>75</GenusId>
    <Name>Oribacterium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>76</GenusId>
    <Name>Oscillibacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>77</GenusId>
    <Name>Oscillospira</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>78</GenusId>
    <Name>Paludibacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>79</GenusId>
    <Name>Papilibacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>80</GenusId>
    <Name>Parabacteroides</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>d968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>81</GenusId>
    <Name>Paraprevotella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>da68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>82</GenusId>
    <Name>Parasutterella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>db68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>83</GenusId>
    <Name>Peptococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>dc68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>84</GenusId>
    <Name>Prevotella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>dd68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>85</GenusId>
    <Name>Proteiniphilum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>de68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>86</GenusId>
    <Name>Pseudobutyrivibrio</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>df68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>87</GenusId>
    <Name>Pseudomonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>88</GenusId>
    <Name>RFN20</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>89</GenusId>
    <Name>Rhodococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>90</GenusId>
    <Name>Rosburia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>91</GenusId>
    <Name>Roseburia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>92</GenusId>
    <Name>Ruminococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>93</GenusId>
    <Name>Schlegelella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>94</GenusId>
    <Name>Schwartzia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>95</GenusId>
    <Name>Selenomonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>96</GenusId>
    <Name>Silanimonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>e968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>97</GenusId>
    <Name>Sphingomonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ea68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>98</GenusId>
    <Name>Spirochaeta</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>eb68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>99</GenusId>
    <Name>Sporobacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ec68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>100</GenusId>
    <Name>Staphylococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ed68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>101</GenusId>
    <Name>Streptococcus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ee68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>102</GenusId>
    <Name>Subdoligranulum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ef68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>103</GenusId>
    <Name>Succiniclasticum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f068b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>104</GenusId>
    <Name>Succinivibrio</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f168b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>105</GenusId>
    <Name>Sutterella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f268b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>106</GenusId>
    <Name>Thermomonas</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f368b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>107</GenusId>
    <Name>Thermotoga</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f468b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>108</GenusId>
    <Name>Treponema</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f568b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>109</GenusId>
    <Name>Turibacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f668b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>110</GenusId>
    <Name>Turicibacter</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f768b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>111</GenusId>
    <Name>Variovorax</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f868b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>112</GenusId>
    <Name>Veillonella</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>f968b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Genus>
    <GenusId>113</GenusId>
    <Name>Alistipes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>a6d31249-45f2-e911-a40b-281878345184</UUID>
  </Taxonomy.Genus>
  <Taxonomy.Kingdom>
    <KingdomId>0</KingdomId>
    <Name>-- N/A --</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>fa68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Kingdom>
  <Taxonomy.Order>
    <OrderId>0</OrderId>
    <Name>-- N/A --</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>fb68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>1</OrderId>
    <Name>Anaeroplasmatales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>fc68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>2</OrderId>
    <Name>Bacteroidales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>fd68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>3</OrderId>
    <Name>Bifidobacteriales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>fe68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>4</OrderId>
    <Name>Cerasiococcales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>ff68b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>5</OrderId>
    <Name>Clostridiales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0069b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>6</OrderId>
    <Name>Coriobacteriales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0169b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>7</OrderId>
    <Name>Desulfovibrionales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0269b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>8</OrderId>
    <Name>Elusimicrobiales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0369b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>9</OrderId>
    <Name>Enterobacterales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0469b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>10</OrderId>
    <Name>Erysipelotrichales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0569b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>11</OrderId>
    <Name>Flavobacteriales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0669b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>12</OrderId>
    <Name>Fusobacteriales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0769b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>13</OrderId>
    <Name>Gastranaerophilales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0869b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>14</OrderId>
    <Name>GMD14H09</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0969b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>15</OrderId>
    <Name>Lactobacillales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0a69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>16</OrderId>
    <Name>Methanomicrobiales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0b69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>17</OrderId>
    <Name>Pasteurellales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0c69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>18</OrderId>
    <Name>Pseudomonadales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0d69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>19</OrderId>
    <Name>RF32</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0e69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>20</OrderId>
    <Name>RF9</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>0f69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>21</OrderId>
    <Name>Rickettsiales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1069b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>22</OrderId>
    <Name>Selenomonadales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1169b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>23</OrderId>
    <Name>Turicibacterales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1269b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Order>
    <OrderId>24</OrderId>
    <Name>Verrucomicrobiales</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1369b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Order>
  <Taxonomy.Phylum>
    <PhylumId>0</PhylumId>
    <Name>-- N/A --</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1469b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>1</PhylumId>
    <Name>Actinobacteria</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1569b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>2</PhylumId>
    <Name>Bacteroides</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1669b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>3</PhylumId>
    <Name>Bacteroidetes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1769b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>4</PhylumId>
    <Name>Cyanobacteria</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1869b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>5</PhylumId>
    <Name>Deferribacteres</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1969b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>6</PhylumId>
    <Name>Elusimicrobia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1a69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>7</PhylumId>
    <Name>Euryarchaeota</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1b69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>8</PhylumId>
    <Name>Fibrobacteres</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1c69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>9</PhylumId>
    <Name>Firmicutes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1d69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>10</PhylumId>
    <Name>Fusobacteria</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1e69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>11</PhylumId>
    <Name>Gammatimonadetes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>1f69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>12</PhylumId>
    <Name>Lachnospiraceae</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2069b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>13</PhylumId>
    <Name>Proteobacteria</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2169b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>14</PhylumId>
    <Name>Spirochaetes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2269b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>15</PhylumId>
    <Name>Tenericutes</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2369b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>16</PhylumId>
    <Name>Verrucomicrobia</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2469b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Phylum>
    <PhylumId>17</PhylumId>
    <Name>Candidatus Saccharibacteria</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>9ef21880-57ee-e911-a40b-28187834307b</UUID>
  </Taxonomy.Phylum>
  <Taxonomy.Species>
    <SpeciesId>0</SpeciesId>
    <Name>-- N/A --</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2569b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>1</SpeciesId>
    <Name>Akkermansia muciniphila</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2669b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>2</SpeciesId>
    <Name>Bacteroides acidifaciens</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2769b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>3</SpeciesId>
    <Name>Candidatus arthromitus SFB</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2869b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>4</SpeciesId>
    <Name>Clostridium cochleatum</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2969b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>5</SpeciesId>
    <Name>Enterobacter arachidis</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2a69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>6</SpeciesId>
    <Name>Lactobacillus intestinalis</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2b69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>7</SpeciesId>
    <Name>Lactobacillus reuteri</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2c69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>8</SpeciesId>
    <Name>Ruminococcus gnavus</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2d69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
  <Taxonomy.Species>
    <SpeciesId>9</SpeciesId>
    <Name>Selenomonas ruminantium</Name>
    <VisibleInUI>true</VisibleInUI>
    <UUID>2e69b104-9c99-e911-abc4-000d3a228e9c</UUID>
  </Taxonomy.Species>
</ExportAllData>